Keratosis pilaris?

Does anyone know a definite cure for KP? I'm sick of it' so sick of it that it is making me depressed

Keratosis pilaris?
Keratosis pilaris, otherwise known as chicken skin ......... little rough bumps which usually appear on the backs of the arms and across the shoulders and upper portion of the body. Tis simply a Vitamin A deficiency..... eat more foods rich in this vitamin ie: carrots, pumpkin, dark green and leafy vegies, eggs, milk and dairy products, margarine and yellow fruits.





This will also solve any night blindness, or sensitivity to bright daylight you may have. Vitamin A will also aid in the proper function of the immune system and protect you from many of the viral and flu infections that are airborne.





CHEERS


♥
Reply:I have had KP since I was a child (on my arms, but has since migrated a little as I age)





One thing that helps (but doesn't cure - there isn't a cure to my knowledge) is exfoliation. I've read that professional chemical peels (done by a dermatologist) can help give longer lasting results, but are not permanent for KP.
Reply:Keratosis pilaris results from a buildup of protein called keratin in the openings of hair follicles in the skin. This produces small, rough patches, usually on the arms and thighs. Though quite common with young children, keratosis pilaris can occur at any age.





Many people are bothered by the goose flesh appearance of keratosis pilaris, but it doesn't have long-term health implications and occurs in otherwise healthy people.





Keratosis pilaris usually resolves without treatment. Dry skin tends to worsen the condition.





No single treatment universally improves keratosis pilaris. But most options, including self-care measures and medicated creams, focus on softening the keratin deposits in the skin.





Prescription medications used to treat keratosis pilaris include:





Ammonium lactate (Lac-Hydrin). Available in a cream or lotion, 12 percent ammonium lactate reduces roughness and softens the keratin plugs. It won't, however, lessen the redness caused by the condition.





Urea (Carmol, Keralac). Urea moisturizes and softens dry, rough skin. It also helps loosen and remove the dead skin cells. Side effects include redness, stinging and skin irritations.





Topical corticosteroids. These anti-inflammatory drugs help decrease cell turnover by suppressing the immune system. Low-potency corticosteroid ointments are usually recommended for sensitive areas such as your face and for treating widespread patches. Doctors usually prescribe corticosteroids for short-term treatment or for temporary relief of symptoms. They aren't used as long-term treatments because of potential side effects.





Topical retinoids. Derived from vitamin A, retinoids work by promoting cell turnover and preventing the plugging of the hair follicle. Retinoids may be an effective treatment, but they can cause bothersome skin irritations, such as severe dryness, redness and peeling. Tretinoin (Retin-A Micro, Avita) and tazarotene (Tazorac) are examples of topical retinoids.





Using a medication regularly may improve the appearance of your skin. But if you stop, the condition returns. And even with medical treatment, keratosis pilaris tends to persist for years.





Be careful with Vitamin A. Vitamin A toxicities in humans may be generally categorised as either acute or chronic. Acute toxicity occurs following ingestion of high doses of vitamin A. It may occur within hours or at most a day or two after a very large intake.





Chronic toxicity occurs after consuming smaller amounts of vitamin A for a long period of time. - several weeks, months or years. Vitamin A has a long biological half-life and it tends to accumulate in the body. The tendency of vitamin A to bio-accumulate suggests that chronic exposure to excessive amounts would be unsafe.





Since vitamin A is stored in the liver, chronic ingestion of megadoses may result in hepatic toxicities. The range of vitamin A doses leading to liver damage vary from 15,000IU per day to 1.4 million IU per day, with an average daily toxic dose of 120,000IU per day.





In patients with renal failure, as little as 4,000IU per day has been known to cause substantial liver damage. Long-term alcohol consumption also enhances liver toxic reactions of vitamin A.





Daily use in pregnant women and those who wish to become pregnant should not exceed 8,000IU per day. Larger amounts taken during pregnancy have been associated with an increased incidence of birth defects. It was found that those who took more than 10,000IU of vitamin A has a higher risk of delivering babies with birth defects compared with women taking 5,000IU.





Bets of luck with this.

palm